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What are the generic names for lasix and prednisone? Do they differ when it comes to how they treat patients with cancer?" Lancet. Published online August 15, 2003. A new study led by Harvard Medical School reports that, in mice, two drugs used to treat certain types of brain tumors can activate a protective mechanism that can extend lifespans. The study was published Sept. 3 by the journal Neuron on behalf of the LongevityDx, a consortium more than 100 neuroscientists and cancer researchers. The consortium was formed in 2009 and is funded by the US Defense Advanced Research Projects Agency (DARPA) with grants to Harvard Medical School and the Howard Hughes Medical Institute. LongevityDx research is published by Nature Publishing Group. It's known that certain types of brain tumors are much more likely to recur than other tumors in patients, and this has led to the development of therapies that kill cancer cells while sparing healthy brain cells. But these agents may also be driving tumor growth by activating an immune response in noncancer cells. The discovery that such a response can extend lifespan in mice has spurred an effort to look for agents that could be harnessed as tools to prevent further Prometrium 200 mg capsule tumor growth, said lead author Mark H. Johnson, Ph.D., the John A. and Elizabeth Mathers Lillian Robert W. Wood Director of Harvard Medical School's Department Brain Sciences. In the new study, Johnson and his colleagues analyzed the mechanism that leads to increased brain cell survival in mice treated with one of these drugs. They identified an immune system component called Th1-cortical cytokine receptor 12 (Nec-1)-driven cell death that causes a drop in immune cell numbers and a corresponding increase in the growth of brain cells, which provides the power for mice to survive. "This mechanism is likely to be important for patients with brain cancer, but we have to determine, experimentally first, whether any of these agents can activate it in people with brain tumors," Johnson said. Previous studies have shown that a specific subset of immune cells called natural killer (NK) can recognize and kill many cancers in animals but do not appear to kill cancer cells in humans. In their new study, Buying dutasteride online Johnson and his colleagues found that treatment with the NK-1 receptor antagonist bortezomib also activates Nec-1-driven cell death and reduces the number of NK cells. The scientists then tested hypothesis that NK cells would need other "killer" to reduce the number of brain cancer cells in mice and showed that activating NK cells by treatment with bortezomib activated Nec-1, while treatment with other NK-cell-blocking drugs did not. "The NK buy generic lasix cells play an important role in maintaining the cancer cells, so when they can't support them and are destroyed, as they in a large proportion of people with brain cancers, that's one of the reasons they don't heal as well and spread. So it's very important to understand exactly who those necessary "killer" cells are," Johnson added. To determine whether Nec-1 activation might be linked to cancer recurrence, the researchers treated mice with a similar agent that inhibits Nec-1. The results showed reduced tumor growth in animals that had been treated with the drug. "If people have tumors, it certainly looks like the immune system is very important in the tumor response," Johnson canada drug pharmacy wichita kansas said. "It may be that they can improve outcomes by changing the way they react against tumors and treating people"
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